In the present study, we first used bioinformatics repositories including Tumor Immune Estimation Resource (TIMER) (http://timer.cistrome.org/) [14], Oncomine (https://www.oncomine.org/) [15] and UALCAN (http://ualcan.path.uab.edu) [16] databases to predict the expression level of ITGBL1 and found that it was upregulated in GC tissues compared with normal tissues. Here, ITGBL1 is linked to neoplasm.