PTK2 and cytomegalovirus infection: The results indicated that HCMV significantly inhibited phosphorylation levels of FAK, AKT, and CAS, and suppressed GTP-Rac through miR-US33-5p level upregulation (Additional file 1: Fig. S1), and that HCMV-miR-US33-5p mimics elicited the same effects as HCMV infection.