ACTA1 and neoplasm: The abundance of certain classical CAF markers (e.g. α‐SMA, fibronectin, periostin, PDGFRβ and paladin) was negligible in CRC cells (epithelial or mesenchymal) in comparison with fibroblasts and (TGF‐β‐stimulated) myofibroblasts (Figure 7b), suggesting that the contribution of CRC cells to the whole tumour expression of these markers is also negligible.