Using Western blotting analysis, we showed that SAHA treatment induced the accelerated phosphorylation signals of LMNA at the Ser390 site, while its acetylation status at K270/311 was declined in different NPC cell lines, including 5–8 F and HNE3 cells (Figures 7A, B). The gene discussed is LMNA; the disease is nasopharyngeal carcinoma.