ILK and esophageal squamous cell carcinoma: In this study, the authors designed rescue experiments to explore the potential mechanism of ILK in affecting the sensitivity of ESCC cells to CDDP chemotherapy by overexpressing β-catenin in ILK-knockdown ESCC cells, then the results of proliferation, migration, invasion, and apoptosis assays showed that the overexpression of β-catenin significantly reversed the ILK knockdown-induced sensitivity of ESCC cells to CDDP chemotherapy.