Given that Hb co‐infection dramatically impaired expulsion of Tm but did not significantly affect canonical drivers (IL‐13) or effectors (goblet cell hyperplasia, increased crypt length, type 2 gene expression) of anti‐Tm immunity, we aimed to identify if Hb was influencing Tm′s ability to establish an infection independent of any effect Hb may exert on adaptive immunity. The gene discussed is IL13; the disease is coinfection.