As our results show, CCL5 and IL-4 were elevated in AA-CM (Ctrl-CM as control) but were suppressed in anti-miR-382+AA-CM (anti-scramble+ AA-CM as control), suggesting that CCL-5 and IL-4 would be the critical mediators in the involvement of miR-382 on AA-induced CKD. The gene discussed is IL4; the disease is chronic kidney disease.