It has been reported that the inhibition of EGFR phosphorylation in A549 cells, overexpressing wild-type EGFR, results in the inhibition of mTOR, which is a key intracellular kinase involved in the regulation of proliferation and cell survival [36,37,38], thus playing a role in NSCLC. The gene discussed is MTOR; the disease is non-small cell lung carcinoma.