Additionally, several non-Wnt molecules, including Enhancer of zeste homolog 2 (EZH2), telomerase reverse transcriptase (TERT), Capillary morphogenesis gene 2 (CMG2), Histone-lysine N-methyltransferase (SMYD3), CD138, and Focal adhesion kinase (FAK), regulate CSCs in TNBC and other cancers via their interaction with Wnt effectors such as β-catenin and LRP6[43-48]. This evidence concerns the gene TERT and cancer.