However, macrophages are also enriched by a keratinocyte-specific INHBA transgene in HPV8-induced skin tumors, and their depletion by anti-CSF1R treatment delayed the onset of tumor formation.18 By contrast, we found that depletion of CSF1R+ TAMs did not reduce INHBA-induced melanoma growth. The gene discussed is CSF1R; the disease is skin neoplasm.