There are a lot of related factors and pathways to the inflammation response in TME, such as tumor macrophages (TAMs), dendritic cells, lymphocytes, proinflammatory cytokines, nuclear factor-κB (NF-κB), and c-Jun N-terminal kinase (JNK), which impacts the progression of the tumor [7]. This evidence concerns the gene NFKB1 and neoplasm.