This suggests two things—(i) that despite shared proximal proximal pathways of immunopathogenesis (i.e., IL15/IL15RA-centric cytokine storm), the immunopathogenesis of KD and MIS-C may diverge distally; and (ii) that IL15/IL15RA, eosinopenia and thrombocytopenia may be inter-related phenomena in the setting of infection and inflammation. The gene discussed is IL15RA; the disease is Thrombocytopenia.