Wang et al. (20) showed that IUGR stimulated jejunum PC and 8-OHdG, and ileum PC, MDA, and H2O2 production, and it decreased the total antioxidant capacity (T-AOC), CAT activity, and glutathione (GSH) content in the jejunum, and CAT activity in the ileum, which suggested that IUGR caused oxidative stress in the intestinal tract. The gene discussed is CAT; the disease is fetal growth restriction.