MYD88 and Sepsis: In this study, we used lipopolysaccharide- (LPS-) treated mice or HL-1 cells to construct a sepsis-induced cardiomyopathy model, observed the miR-340-5p and MyD88 expression levels, detected the influence of miR-340-5p overexpression or knockdown and MyD88 siRNA on the SIC model, and explored its specific mechanism.