TGFBR1 and acute respiratory distress syndrome: In the context of ARDS, exosomes derived from the adipose tissue of lean mice, which were pretreated with miR-122-5p agomir, significantly mitigated lung injury, fibrosis, and ROS accumulation (Figures 6(b) and 6(d)), improved oxygenation and the free GSH concentration (Figures 6(e) and 6(j)), attenuated hyperpermeability (Figures 6(f)–6(h)), and decreased the collagen levels and TGFβ-R1 expression in the lung tissue (Figures 6(i) and 6(k)).