Current understanding of diet protein’s participation in MetS intervention focuses on its effect on alteration of glycemic response and glycemic load, by delaying gastric emptying and stimulating insulin, glucagon-like peptide-1, and peptide YY secretion, and outcomes in preventing specific MetS components via fat mass reduction while maintaining lean body mass during energy-deficiency and increasing HDL cholesterol (Chalvon-Demersay et al., 2017). Here, GCG is linked to metabolic syndrome.