STING1 and vasculitis: In 2014, mutations in transmembrane protein 173 (TMEM173) (the gene which encodes STING) were found to increase production of IFN‐β in PBMCs and fibroblasts from pediatric patients presenting with recurrent fevers, ulcerative skin lesions, vasculitis, and interstitial lung disease, in addition to systemic inflammation, cutaneous vasculopathy, and pulmonary inflammation [70, 71].