IL1B and endometritis: The endometritis features are inflammation, tissue impairment, and necrosis, which are developed and maintained under the expression of inflammatory mediators such as prostaglandin E2 (PGE2), nitric oxide (NO), interleukin-1β (IL-1β), interleukin-6 (IL-6), interleukin-8 (IL-8), and damage-associated molecular patterns (DAMPs).