The second report [2] supports the above with 11 in a cohort of 187 hospitalized patients with laboratory confirmed COVID-19, of which 27.8% (n = 52) revealed myocardial damage noted by elevated troponin T (TnT) levels, providing additional novel insights concerning levels of C-reactive protein and N-terminal pro-B-type natriuretic peptide (NT-proBNP). The gene discussed is CRP; the disease is COVID-19.