As the string networks of the upregulated proteins in MiGn and MiGa (Supplementary Figures S61 and S62) show, albumin/apolipoprotein-centered interactions in MiGn lose importance in MiGa and give way to, among others, members of the glutathione antioxidant system (glutathione S-transferase theta-1 (GSTT1), glutathione peroxidase 3 (GPX39)); these disturbances are implicated in tumor initiation and progression [44]. This evidence concerns the gene GPX3 and neoplasm.