Our previous study also found that DIP unregulated the expression of Bax and caspase-3 genes and reduced Bcl-2/Bax heterodimer formation, regulated the HCC-LM3 cell cycle, and promoted the role of the cellular mitochondrial apoptotic pathway, suggesting that DIP has potential therapeutic value for liver injury [16]. Here, CASP3 is linked to hepatocellular carcinoma.