Cyanidin-3-O-glucoside (C3G) prevented the progression of allergic asthma in a murine model through STAT6/GATA3 signaling suppression [34], whereas another cyanidin, procyanidin A2, reduced the expression of CCL26 in human keratinocytes via JAK1/STAT6 signaling [35]. The gene discussed is CCL26; the disease is allergic asthma.