The authors showed that although weak and focal expression was seen in AIP and a normal pancreas, PDAC was characterized by strong overexpression of Maspin, IMP3, and S100P in 95%, 75%, 75%, respectively, whereas weak and no expression was seen in 0%, 25%, 10% of PDAC cases, respectively. The gene discussed is S100P; the disease is autoimmune pancreatitis.