We found somatic alterations consistent with the literature, such as AR, TP53, FOXA1, SPOP, PTEN, or ATM alterations in PC [23], and TP53, KDM6A, RB1, ATM, FAT1, or ARID1A alterations in bladder urothelial cancer [7]. This evidence concerns the gene AR and bladder transitional cell carcinoma.