Interestingly, KLC2 and LINC00152 were significantly upregulated in HCC compared to the surrounding non-tumor liver tissue in the TCGA dataset (LIHC cohort [4,17]: KLC2 5.73 ± 0.09 (mean ± SEM) (SL) vs. 7.51 ± 0.05 (HCC), p < 0.001, Figure 6C; LINC00152 7.51 ± 0.19 (mean ± SEM) (SL) vs. 10.22 ± 0.07 (HCC), p < 0.001, Figure 6D), while miR-143a-3p expression levels were significantly decreased in HCC compared to normal liver tissue in this cohort (15.76 ± 0.09 (mean ± SEM) (SL) vs. 15.14 ± 0.06 (HCC), p < 0.001, Figure 6E). This evidence concerns the gene KLC2 and neoplasm.