Although many aspects of endometriosis onset and development still remain unclear, continuous research advances highlight the critical role of inflammation and TGFβ in growth, cell adhesion, endometrial epithelial-mesenchymal transition (EMT) and invasion, angiogenesis, fibrogenesis and immune escape, both in OvCa and endometriosis [7]. Here, TGFB1 is linked to endometriosis.