Autologous injections of mADSCs into T2D model mice have been found to result in increased insulin sensitivity as marked by a decrease in glucose plasma levels, a decrease in adipocyte size, a restoration of β cell mass, and a reduction in pro-inflammatory insulin desensitization markers, such as IL-6, TNF-α, and F4/80 in the liver. This evidence concerns the gene INS and type 2 diabetes mellitus.