As mentioned above, there was a diversified regulation of miR-128 levels related to the oxidative stress mechanism in AD, PD, and HD [212], involving the TrkC.T1 receptor and the TNF-α level in astrocytes in ALS [213], and regulation of pleiotropic cytokine TGFβ related to T-helper 17 (Th17) cells in immunological effects in MS [214]. The gene discussed is TNF; the disease is Alzheimer disease.