Moreover, apoptosis and inflammation-associated events within the encephalon of PD-experiencing individuals are corroborated by numerous episodes, encompassing elevated quantities of the p53 gene (tumor suppressor), an inflammatory mediator denominated as interferon-gamma (IFN-γ), NF-ΚB, caspase activation within the SN, and alteration in the proapoptotic gene operation [96,97,98,99]. This evidence concerns the gene IFNG and Parkinson disease.