Other molecular targets thathave been demonstrated to be important for ALS onset and progression are superoxide dismutase (SOD1) [16] and DNA/RNA-binding protein FUS/TLS (FUused in Sarcoma/Translocated in LipoSarcoma, also called “FUS”) [17], which appear to be mutated in the patients. Here, SOD1 is linked to amyotrophic lateral sclerosis.