Targeting ALDH3A1 in gastric cancers has anti‐cancer effects, and prevented 40% of NADH generation, leading to the conclusion that a large source of β‐oxidation of fatty acids is dependent on ALDH3A1,47 and placing ALDH at a unique position of reducing genotoxic effects, altering survival, and providing a fuel source through β‐oxidation in EAC cells. Here, ALDH3A1 is linked to gastric cancer.