Next, we tried to test if WNT10B was responsible for the upregulated expression of IFNG and IL17A in PTSD, and consequently, mimicked the upregulated level of WNT10B by adding recombinant human WNT10B into the culture medium consisting of pre-activated PBMCs from healthy donors and compared the level of IFNG and IL17A. This evidence concerns the gene IL17A and post-traumatic stress disorder.