In another research on a model of myocardial ischemic injury, exosomes from BM-derived mesenchymal stem cells reduced apoptosis and the size of myocardial infarction and, after that, recovered heart function by persuading cardiac autophagy via two pathways such as AMPK/mTOR and Akt/mTOR routes [55]. This evidence concerns the gene MTOR and myocardial infarction.