For example, an exploration in hepatocellular carcinoma indicated that miR-383-5p constrained cell growth by regulating AKR1B10 expression.24 Another document in lung cancer implied that miR-383-5p regulated lung cancer cell behaviors by targeting CIP2A.25 In the current study, miR-383-5p was declined in CRC and validated to be sponged by hsa_circ_0000467. This evidence concerns the gene AKR1B10 and lung carcinoma.