Moreover, we analyzed tumor tissues by an IHC assay and found that RBMS1-depletion induced reduction of PD-L1 and elevated infiltration of CD8+ T cells could be reversed by re-expression of B4GALT1, resulting in increased levels of PD-L1 and Ki67, but decreased infiltration of CD8+ T cells (Fig. 6G). The gene discussed is CD8A; the disease is neoplasm.