SQSTM1 and B-cell non-Hodgkin lymphoma: The miR-17/92 cluster was also found overexpressed in B-cell Lymphomas, and the individual members miR-19a/19b were required and sufficient for the B-cell lymphoma tumorigenesis [20, 21].The study performed by Meenhuis A found miR-17/20 in combination with two other miRNAs promoted expansion and replating capacity of myeloid progenitors by targeting sequestosome 1–regulated pathways [22].