IL-18 and IL-1β were selected as the inflammation indicators in this study; both are strongly linked to the onset and progress of COPD, in which IL-18 is an important class of proinflammatory factors that can induce macrophage and lymphocyte infiltration and accelerate vascular remodeling, while IL-1β can activate the MAPK signaling pathway, thereby increasing the expression of a series of inflammation-related factors [10] and aggravating the condition of patients. Here, IL1B is linked to chronic obstructive pulmonary disease.