CXCR4 and cancer: Consistently with these results, our group also proved the T22-PE24-H6 capacity to block dissemination in a diffuse large B-cell lymphoma (DLBCL) model without associated toxicity, by the elimination of CXCR4+ cancer cells through apoptosis induction (Falgàs et al., 2020), a result consistent with the pharmacokinetics of T22-P26-H6 that show a fast biodistribution in the bloodstream followed by a slow elimination phase having a half-life of 30 hours (Céspedes et al., 2007).