It is known that Gas6 binds with the strongest affinity to Axl and to a lesser extent to Tyro3 and MerTK: while acting as immune regulator, the Gas6 activation finally results in dampening Toll-like receptors inflammatory signaling [13, 19, 20, 22, 23] thus counteracting inflammatory responses induced also by pathogen infections [9, 20]. The gene discussed is GAS6; the disease is infection.