Tang et al. revealed that LINC00115, which is activated by TGF-β, promotes GSC tumorigenicity by augmenting ZNF596 transcription via preventing binding of miR-200s to the 5′-UTR of ZNF596, thereby promoting ZNF596/EZH2/STAT3 signaling axis and GBM tumorigenesis (Yang et al., 2020a). Here, ZNF596 is linked to glioblastoma.