Zhao et al. (2019) reported that ZEB1 transcriptionally activated SNHG14, which displays immune evasion effects by inhibiting cytotoxic cell activation (Figure 4). ROR1-AS1 was found to be packaged into exosomes and derived from tumor cells. Functional analysis suggested that exo-ROR1-AS1 facilitates GBM development through sponging miR-4686 (Chai et al., 2020). The gene discussed is ROR1; the disease is glioblastoma.