This study predicted the Toll-like, MAPK, cytokine-cytokine receptor interaction, ErbB pathways (ALV-J induced tumor livers vs. normal livers) and found that lncRNAs with m6A modifications were common in tumor livers induced by ALV-J infection; hence, we suspected that lncRNAs with m6A modifications might mediate tumor formation and mediate viral immunosuppression. Here, EGFR is linked to neoplasm.