For ADAM10, our results demonstrate a function at a later stage in COVID‐19 pathogenesis, i.e., in syncytia formation of lung cells, which is one pathological hallmark in the lungs of COVID‐19 patients and assumed to occur through the fusion of infected cells expressing the S protein with cells expressing its receptor ACE2 (Bussani et al, 2020; Braga et al, 2021). The gene discussed is ACE2; the disease is COVID-19.