Previous studies have demonstrated that transcriptional and post‐transcriptional suppression of SLC7A11 by transcription factors (such as ATF3 and ATF4), H2A deubiquitinases (such as BAP1), and epigenetic modifications (such as H2Bub1) can promote erastin‐induced tumour cell ferroptosis.40, 41, 42, 43. The gene discussed is SLC7A11; the disease is neoplasm.