Keratin 14 (K14), which contains a nonhelical tail domain involved in K5–K14 large bundles self‐organising, has been shown to be overly expressed in basal‐like bladder cancer, and K14+ cell subpopulations serving as bladder cancer stem cells contribute to tumourigenesis and chemoresistance in vivo.32, 33, 34. This evidence concerns the gene KRT5 and urinary bladder carcinoma.