FUT4 and bronchopulmonary dysplasia: Furthermore, administration of Fut9 had increased SSEA-1 expression and cell proliferation resulting in the reverses of hyperoxia-induced cell death, airway inflammation, and lung injury in the murine model of neonatal bronchopulmonary dysplasia, suggesting the roles of SSEA-1 in immunomodulation and tissue regeneration (Chaubey et al., 2021).