For instance, M1-MG may exhibit excessive phagocytosis, also called phagoptosis (Wang K. et al., 2021), eliminating viable neurons (Nehera et al., 2013; Brown and Neher, 2014) and live synapses (Shi X. et al., 2021) through multiple epidermal growth factor-like domains protein 10 (MEGF10) and tyrosine-protein kinase Mer (MERTK) in the post-stroke repair and remodeling stage. This evidence concerns the gene MERTK and Stroke.