Additional analyses revealed that IκBα-MnNP treatment significantly increased the percent of classical (M1-like) monocytes (F4/80int/Ly6C+/Ly6G-) in the ascites and significantly increased the percent of NK cells (CD3−/B220−/NKp46+) in the tumor (Fig. 6H, I). The gene discussed is PTPRC; the disease is neoplasm.