AAV9-IL9 showed remarkable enrichment over all other interleukins in both Pool 60 (Fig. 1B) and Pool 15 (Fig. 1C), and in combination with IL3, IL4, IL13, and IL15 (Pool 5), significantly improved CVB3-induced myocarditis (Figs. 2 and 5). This evidence concerns the gene IL4 and myocarditis.