ADAMTS10 and acromelic dysplasia: Recessive ADAMTS10 mutations lead to an acromelic dysplasia, Weill-Marchesani syndrome 1 (WMS1) (Dagoneau et al., 2004), whereas dominant FBN1 mutations cause a phenotypically similar disorder, WMS2 (Faivre et al., 2003b), suggesting a functional relationship between ADAMTS10 and fibrillin-1 (Hubmacher and Apte, 2015; Karoulias et al., 2020).