inhibited AST and ALT activities; attenuated total glutathione S‐transferase activity (GST); enhanced superoxide dismutase (SOD) and catalase (CAT) activities; attenuated liver fibrosis, decreased expression of α‐smooth muscle actin (α‐SMA), induced expression of active matrix metalloproteinase‐2 (MMP2), and inhibited TIMP2 level. The gene discussed is ACTA1; the disease is Hepatic fibrosis.